Congratulations on the book launch! The Hunger Code is an apt title, because the real problem most people are trying to solve isn’t “willpower,” it’s decoding what their appetite signals are actually responding to.
What I appreciate in your work (and what I hope this book continues) is the consistent reframing of hunger as biology + environment, not moral failure. When someone understands the different “hunger channels” (homeostatic, hedonic, conditioned/social), they stop fighting themselves and start designing around the signal: meal composition that actually satisfies, fewer engineered cues, and routines that don’t keep the brain in constant negotiation.
Also: launching a book like this right now feels timely. In an ultra-processed food environment, hunger gets hijacked, not because people are weak, but because the stimulus environment is aggressive. A book that helps readers recognize the difference between “my body needs fuel” and “my brain is being prompted” can genuinely change outcomes.
I’ve seen you post about various podcast interviews you’ve done in the lead up to today. It would be fantastic if you could post links to the pods when they’re released 🙏🏻
Congrats dear colleague! As an oncologist, I love your work and read “cancer code” several times. Can’t wait to read your new book and share the advices with my patients
My only disappointment is: the only Hunger Code audiobook I could find was through Amazon’s Audible. This platform does not allow for a one-time book purchase that I can return to for future reads. I’ve gone back to my Apple iBooks from you and miss that.
One question I keep returning to is whether we implicitly assume that everyone starts with the same beta cell mass and reserve.
Remission of type 2 diabetes is clearly possible in many individuals — and challenging the idea of inevitable progression was enormously important. But biological variability in beta cell reserve may be far greater than we currently acknowledge.
The critical moment, in my view, is not when diabetes is diagnosed — but when glucose regulation first begins to struggle. At that point, differentiation matters.
Is this primarily an overload problem driven by insulin resistance?
Or is it, in some individuals, a limited or delayed insulin response from the start?
If we assume uniform pancreatic capacity and interpret every early dysregulation as resistance, we risk applying the wrong mechanical solution to the wrong biological architecture. In certain physiological systems, that could mean intensifying fasting, pushing substrate restriction, or focusing solely on lowering glucose — when the underlying issue may be constrained beta cell reserve or impaired timing.
Physiologically, overload and limited reserve are not the same system.
The numbers may look similar — but the mechanics are different.
Congratulations! Thank you for doing your part to heal our sick population!!
Congratulations on the book launch! The Hunger Code is an apt title, because the real problem most people are trying to solve isn’t “willpower,” it’s decoding what their appetite signals are actually responding to.
What I appreciate in your work (and what I hope this book continues) is the consistent reframing of hunger as biology + environment, not moral failure. When someone understands the different “hunger channels” (homeostatic, hedonic, conditioned/social), they stop fighting themselves and start designing around the signal: meal composition that actually satisfies, fewer engineered cues, and routines that don’t keep the brain in constant negotiation.
Also: launching a book like this right now feels timely. In an ultra-processed food environment, hunger gets hijacked, not because people are weak, but because the stimulus environment is aggressive. A book that helps readers recognize the difference between “my body needs fuel” and “my brain is being prompted” can genuinely change outcomes.
Wishing you a huge release week!
"...the consistent reframing of hunger as biology + environment, not moral failure." YES!! 🎯✅💯
Congratulations! I can’t wait to read it.
I’ve seen you post about various podcast interviews you’ve done in the lead up to today. It would be fantastic if you could post links to the pods when they’re released 🙏🏻
I just purchased your book on Amazon, and I can't wait to read it. You might also enjoy my website: https://carbsyndrome.com/
Congrats dear colleague! As an oncologist, I love your work and read “cancer code” several times. Can’t wait to read your new book and share the advices with my patients
This is working already. So simple. Will have questions for the group talk.
You need to update your cancer code book.
It actually annoyed me no end that you gave Thomas Seyfried zero credibility to the metabolic theory of cancer.
Treating cancer as a genetic disease is false.
Please FIX it.
È disponibile in italiano
My only disappointment is: the only Hunger Code audiobook I could find was through Amazon’s Audible. This platform does not allow for a one-time book purchase that I can return to for future reads. I’ve gone back to my Apple iBooks from you and miss that.
my book was in the mailbox today - already diving in. Love all your books!
I have recommended the obesity code to hundreds of people. I look forward to reading this new book!
One question I keep returning to is whether we implicitly assume that everyone starts with the same beta cell mass and reserve.
Remission of type 2 diabetes is clearly possible in many individuals — and challenging the idea of inevitable progression was enormously important. But biological variability in beta cell reserve may be far greater than we currently acknowledge.
The critical moment, in my view, is not when diabetes is diagnosed — but when glucose regulation first begins to struggle. At that point, differentiation matters.
Is this primarily an overload problem driven by insulin resistance?
Or is it, in some individuals, a limited or delayed insulin response from the start?
If we assume uniform pancreatic capacity and interpret every early dysregulation as resistance, we risk applying the wrong mechanical solution to the wrong biological architecture. In certain physiological systems, that could mean intensifying fasting, pushing substrate restriction, or focusing solely on lowering glucose — when the underlying issue may be constrained beta cell reserve or impaired timing.
Physiologically, overload and limited reserve are not the same system.
The numbers may look similar — but the mechanics are different.